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Finally, dihydrolipoamide dehydrogenase activity in the glycine cleavage system (L protein) and in the -ketoacid dehydrogenase complexes (protein E3) catalyzes the conversion of the dihydrolipoyl moiety (reduced form) to the lipoyl moiety (oxidized form)

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Iron toxicity and hepatic fibrogenesis Iron toxicity Abnormal iron homeostasis in HFE-hemochromatosis can lead to a wide variety of different iron-induced pathologies, including arthritis, cardiomyopathy, diabetes, cirrhosis, and HCC

A small amount of APAP is oxidized by the enzyme CYP2E1 to NAPQI, a toxic metabolite that, under normal circumstances, is quickly rendered nontoxic through an interaction with hepatic glutathione (GSH)